divided structures, and reactionary inflammation is not of sufficient extent to enhance prompt repair. In fact, some cases of this kind seem to progress more favorably, when no attempt at immobilization of the affected member is attempted.

If some freedom of movement is allowed, acute inflammation resulting in nature's provisional swelling soon develops and repair is hastened because of increased vascularity. But where luxation of phalanges accompanies sprain, reposition and immobilization are necessary—that is if cases are thought likely to benefit by any treatment.

Luxations—Dislocations.

Luxation or dislocation is a condition where the normal relation between articular ends of bones has been deranged to the extent that partial or complete loss of function results. When a bone is luxated (out of joint), there has occurred a partial or complete rupture of certain ligaments or tendons; or a bone may be luxated when an abnormal or unusual elasticity of inhibitory ligaments or tendons obtains.

Luxations may be practically classified as temporary and fixed. In temporary luxations, disarticulation is but momentary and spontaneous reposition always results; while a fixed luxation does not reduce spontaneously but remains luxated until reposition is effected by proper manipulation and treatment. Fixed luxation may be of such character as to be practically irreducible because of extensive damage done to ligaments or cartilage. Where a complete luxation of the metacarpophalangeal joint exists, it is probable that in most cases sufficient injury to collateral and capsular ligaments has been done to render complete recovery improbable, if not impossible.

Temporary luxation of the patella is a common affection of the horse and fixed luxation of this bone also occurs. As a matter of fact, in the horse, patellar luxation is the one frequent affection of this kind.

As a rule, complete disarticulation immobilizes the affected joint and in most instances there is noticeable an abnormal prominence in the immediate vicinity—in patellar luxation, the whole bone. In other instances the articular portion only, of the affected bone is malpositioned. Usually, luxation and fracture may be differentiated in that there is no crepitation in luxation and more or less crepitation exists in fracture.

It is evident, when one considers the symptomatology and nature of the affection, that fixed luxation is usually caused by undue strain or violent and abnormal movement of a part. Joints having the greater freedom of movement are apt to suffer luxation more frequently.

Arthritis.

The study of arthritis in the horse is limited to a consideration of joint inflammations which, for the most part, are of traumatic origin. Unlike the human, the horse is not subject to many forms of specific arthritis—tubercular, gonorrheal, syphilitic, etc.

A practical manner of classification of arthritis is traumatic and metastatic.

Traumatic arthritis may result from all sorts of accidents wherein joints are contused. Such cases may be considered as being caused by direct injuries. Instances of this kind, depending on the degree of insult, manifest evidence of injury which ranges from a simple synovitis to the most active inflammatory involvement of the entire structure and adjacent tissues.

The reactionary inflammation which attends a case of tarsitis caused by a horse being kicked is a good example of the result of direct injury. Such cases, if the contusion is of sufficient violence, result in arthritis and periarthritis. In inactive farm horses, during cold weather, this condition becomes chronic, swelling remains for weeks after all lameness and pain have subsided and occasionally hyperthrophy is permanent.

Arthritis occasioned by indirect injury, such as characterizes joint inflammation from continuous concussion, is seen in horses that are worked at a rapid pace on city streets or other hard road surfaces. Such affections may be acute, as in some cases of spavin, but are usually inflammatory conditions that do not occasion serious disturbance when these affections become chronic. If the involvement persists with sufficient active inflammation, there may follow erosion of cartilage and incurable lameness. If extensive necrosis of cartilage takes place, the attendant pain will be sufficient to cause the animal to favor the diseased part and such immobilization enhances early ankylosis—nature's substitute for resolution in this disease.

Wounds invading the tissues adjacent to joints, when these wounds are of considerable extent, cause inflammation of such articulations by contiguous extension of inflammation. As long as an injury remains practically aseptic, or if infected and the septic process does not involve the joint proper by direct extension, no more serious disturbance than a simple synovitis will result. If, instead, a periarthritic inflammation is serious or destructive in character, the type of arthritis will be grave—even though due to an indirect cause.

Where a vulnerant body penetrates all structures and invades the interior of the joint capsule the result is that a more or less active disturbance is incited. The introduction of a sterile instrument into a joint cavity, under strict asepsis, where a perfect technic is executed, does not cause perceptible manifestation of the injury, if the opening so made is small—such as a suitable exploratory trocar makes. But a puncture made in a similar manner and with the same instrument without due regard to asepsis is likely to cause an infectious synovitis and arthritis usually follows.

A larger opening than is produced by means of an exploratory trochar may be made into a joint cavity, causing escape of synovia as it is secreted for days and even for weeks and no serious or permanent trouble is experienced in some cases. If the synovitis or arthritis remains non-infected and the wound, traumatic or surgical, is not too large, healing by granulation occurs, and the discharge of synovia ceases. However, if synovial discharge persists too long because of tardy closure of an open joint, there is great danger of infection gaining entrance into the synovial cavity, or in some instances, desiccation of endothelial cells of the articulation occurs, in areas, and the reactionary inflammation eventually results in ankylosis.

A small puncture which introduces into the synovial cavity infectious material of active virulence will cause an arthritis that is more serious, much more painful and more difficult to handle than is occasioned by a wound of moderate size, that affords ready escape of synovia even through the virulence of the infection be the same.

Synovia is a good culture medium and the environment is ideal for multiplication of bacteria; consequently, the grave disturbances which may attend the introduction of pathogenic organisms into a synovial cavity as the result of a puncture wound are not to be forgotten. The veterinarian is in no position to estimate the virulency of organisms so introduced; neither can he determine the exact degree of resistance possessed by the subject in any given case. Therefore, he is uncertain as to the best method of handling such cases where an injury has been recently inflicted and positive evidence of the existence of an infectious synovitis is not present. If one could determine in advance the degree of infection and injury that is to follow small penetrant wounds of joint capsules, it would then be possible to select certain cases and immediately drain away all synovia and fill the cavity by injection with suitable antiseptic solutions.

This offers a broad field for experimentation which will in time be productive of a radical change in the manner of treating such cases.

Metastatic arthritis is seen more frequently in colts or young